Patients usually
receive EECP treatment for 35 hours divided into one or two
60-minute treatment sessions five days per week. Patients who
undergo two treatment sessions in one day require rest between
sessions. EECP treatment uses unique equipment to inflate and
deflate a series of pneumatic compressive cuffs around the lower
extremities. Treatment is administered on a padded table where three
sets of electronically controlled inflation and deflation valves are
located. These valves are connected to specially designed adjustable
cuffs that are wrapped firmly, but comfortably around the patient's
calves, lower thighs, and upper thighs, including the buttocks. The
design of the cuffs permits significant pressure to be applied to
the arteries and veins at relatively low air pressures. Running
electrocardiogram signals through a microprocessor that monitors the
treatment process regulates timing for inflation and deflation.
While the heart is
at rest the cuffs are inflated in rapid sequence from the calves
upward, creating a pressure wave that increases diastolic pressure,
coronary artery perfusion pressure, and blood flow to the heart
muscle. This compression of the blood vessels in the legs also
increases the volume of blood returned to the right side of the
heart via the venous system.
Instantaneous
deflation of all cuffs at the onset of the heart's contraction
lowers the resistance the heart must pump against, decreasing the
heart's workload.
This latter effect,
when coupled with increased venous return, significantly raises
cardiac output. The overall effect is to increase the oxygen supply
of the heart, while decreasing its oxygen demand. Significant
obstruction in one or more coronary arteries can create a pressure
difference between areas of the heart muscle that receive and those
that do not receive enough blood. Repeated and pulsed increases in
pressure during diastole may stimulate opening or formation of
collateral channels across this pressure gradient within the heart
muscle, resulting in increased blood supply to deprived tissues. The
body has its own solution to an inadequate blood supply caused by
blocked or partially blocked arteries. When an artery is severely
narrowed, the body can increase the amount of blood flowing to the
heart muscle by opening up small branches of nearby arteries.
Known as collateral
circulation, these networks of blood vessels create new routes for
blood to detour around clogged arteries.
EECP treatment can
reduce or eliminate the frequency and intensity of chest pain,
decrease the need for medication, and greatly improve the ability to
participate in activities of daily living. After receiving EECP
treatment, patients often are able to enjoy moderate exercise for
the first time since developing angina.
The development of
a collateral circulation is particularly important in the heart
muscle where it may be life saving. However, the development of
collateral circulation is a gradual process, and not everyone has
the same ability to develop these networks. EECP treatment appears
to stimulate the natural process of developing collateral
circulation, but there are probably other, yet unexplained,
mechanisms that contribute to the long-lasting effects of EECP
treatment.
What Other Conditions Benefit From
EECP?
EECP isn’t just a powerful therapy
for angina. Virtually any condition in which poor circulation is a
contributing factor can benefit from EECP. Here is just a partial
list:

Congestive heart
failure
Diabetic neuropathy
Erectile dysfunction
Hypertension
Kidney disease
Memory disorders
Peripheral vascular disease
Parkinson’s disease
Vision problems
Research Studies & Demonstrated
Results
Angina
An international study group
evaluated EECP’s effectiveness for patients with chronic stable
angina at seven medical centers. After treatment, 85 percent of
patients experienced a significant improvement in their angina
symptoms, and 83 percent showed a measurable increase in blood flow
to the heart. Exercise capacity also improved significantly.
American Journal
of Cardiology, 2002; 89(7): 822-4.